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ISSN: 1001-0602
EISSN: 1748-7838
2020 impact factor 25.617*
(Clarivate Analytics, 2021)
About the cover
The unique autophagy mechanism in RAS-mutated cancer cells is depicted, with glowing dots representing activation of RAS-induced non-canonical autophagy via ATG8ylation (RINCAA) and PI4KB S256/T263 phosphorylation. Five multilaminar structures illustrate RAS-induced multivesicular/ multilaminar bodies of ATG8ylation (RIMMBA), which support tumor survival. Light beams symbolize the therapeutic potential of RINCAA-targeting strategies. Art design by Rui Xie. See page 399–422 by Xiaojuan Wang et al. for details.
RESEARCH HIGHLIGHTS
FUSTer than stress granules: a prion-like domain warns plants of heatCORRIGENDA
Author Correction: A thermosensor FUST1 primes heat-induced stress granule formation via biomolecular condensation in ArabidopsisEditorial Expression of Concern
Editorial Expression of Concern: p53’s mitochondrial translocation and MOMP action is independent of Puma and Bax and severely disrupts mitochondrial membrane integrityRESEARCH HIGHLIGHTS
Targeting an alternative route: autophagy in RAS-driven cancersORIGINAL ARTICLES
Oncogenic RAS induces a distinctive form of non-canonical autophagy mediated by the P38-ULK1-PI4KB axisLETTERS TO THE EDITOR
Mechanisms of chromatin remodeling by the human Snf2-type ATPase SNF2HCORRIGENDA
Author Correction: Improved survival of porcine acute liver failure by a bioartificial liver device implanted with induced human functional hepatocytes
